Glial Profiling Panel

Helping Your Research

Comprehensively study the role of astrocytes, microglia, and oligodendrocytes in health and disease with the nCounter Glial Profiling Panel. Decipher the complex interplay between glial cells, peripheral immune cells, and neurons that underlies neurodegenerative & neuroinflammatory disorders and neurotrauma such as stroke, spinal cord injury, and traumatic brain injury.

Medallion for Glial Profiling panel

How It Works

The Glial Profiling panel covers comprehensive glial cell biology involved in both homeostasis and disease and can be run on its own or paired with the Neuropathology or Neuroinflammation panels.


Profile 770 human or mouse genes across 50+ pathways involved in glial cell biology:

  • Cell stress & Damage Response
  • Pathways Regulating Glia
  • Inflammation & Peripheral Immune Invasion
  • Glial Cell Homeostasis & Activation
  • Neurotransmission

Quantify the relative abundance of 5 CNS cell types and 14 peripheral immune cells


Customizable with Panel Plus option – add up to 55 genes of your choice

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Product Information

Product Specifications
Functional Annotations
Cell Type Gene Coverage
Catalog Information
Product Specifications
Functional Annotations
Cell Type Gene Coverage
Catalog Information

Related Resources

See All Resources
Product Bulletin Glial Panel – Product Bulletin
Flyer Glial Cell Publications – Flyer
Blog Post Rediscovering Microglia: Dr. Oleg Butovsky Discusses the Untapped Potential of Microglia in Neurodegenerative Diseases
Blog Post The Neuroinflammatory Puzzle of the Autoimmune Disease Multiple Sclerosis
Manual/Instructions NanoU Training Videos


View All Publications

Spatial transcriptomics reveals molecular dysfunction associated with cortical Lewy pathology

A key hallmark of Parkinson’s disease (PD) is Lewy pathology. Composed of α-synuclein, Lewy pathology is found both in dopaminergic neurons that modulate motor function, and cortical regions that control cognitive function.

Digital spatial profiling of segmental outflow regions in trabecular meshwork reveals a role for ADAM15

In this study we used a spatial transcriptomics approach to identify genes specifically associated with either high or low outflow regions in the trabecular meshwork (TM) that could potentially affect aqueous humor outflow in vivo. High and low outflow regions were identified and isolated from organ cultured human anterior segments perfused with fluorescently-labeled 200 nm FluoSpheres.

Clinically relevant molecular hallmarks of PFA ependymomas display intratumoral heterogeneity and correlate with tumor morphology

Posterior fossa type A (PF-EPN-A, PFA) ependymoma are aggressive tumors that mainly affect children and have a poor prognosis. Histopathology shows significant intratumoral heterogeneity, ranging from loose tissue to often sharply demarcated, extremely cell-dense tumor areas.

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